Animal StudyPubMed ID: 20217303·2010

Semax Effects on BDNF, NGF, and GDNF: Neurotrophic Factor Signaling in Recovery From Neuronal Injury

Kamayev YA, Ashmarin IP, Lyapina LA.

Journal of Molecular Neuroscience, 2010

Key finding

Semax increased BDNF mRNA 3.4-fold, increased NGF mRNA 2.6-fold, increased GDNF mRNA 2.1-fold in post-stroke brain.

Summary

Study measuring semax effects on neurotrophic factor expression (BDNF, NGF, GDNF mRNA) in injured and uninjured brain regions.

Read the study

The full citation, abstract, and (when available) the full text can be accessed on PubMed. PeptideMark summaries are condensed overviews — we encourage researchers to read primary sources.

View on PubMed

Related compound

SemaxL3

A synthetic peptide analog of ACTH(4-10) approved in Russia as a nootropic, studied for cognitive enhancement and neuroprotection.

More research on Semax

Semax Restores Behavioral Performance and Spatial Memory in Alzheimer Disease Model Mice

Neurobiology of Aging · 2011 · Animal Study

Semax Reduces Brain Inflammation and Microglial Activation After Stroke

Journal of Neuroimmunology · 2010 · Animal Study

Semax Improves Optic Nerve Function and Visual Acuity in Patients with Optic Neuropathy

Journal of the Neurological Sciences · 2009 · Human RCT

Semax Effects on Immune Function: Enhanced T-Cell Proliferation and Altered Cytokine Profile

International Immunology · 2009 · Human Pilot

Nasal Peptide Semax: Tolerability Profile and Safety Data Across Multiple Clinical Studies

Drug Safety · 2009 · Review